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RELATION BETWEEN HEPATITIS B CARRIER STATUS AND ANTIBODY AGAINST SYNTHETIC PLASMODIUM FALCIPARUM ERYTHROCYTE SURFACE (PF155 - RESA) ANTIGEN
Affilliation
Universidade Federal de Mato Grosso do Sul. Faculdade de Ciências Médicas. Núcleo de Estudos de Doenças Infecciosas e Tropicais. Cuiabá, MT, Brasil.
Universidade Federal de Mato Grosso do Sul. Faculdade de Ciências Médicas. Núcleo de Estudos de Doenças Infecciosas e Tropicais. Cuiabá, MT, Brasil.
Fundação Oswaldo Cruz. Instituto Oswaldo Cruz. Departamento de Virologia. Rio de Janeiro, RJ, Brasil.
Universidade Federal de Mato Grosso do Sul. Faculdade de Ciências Médicas. Núcleo de Estudos de Doenças Infecciosas e Tropicais. Cuiabá, MT, Brasil.
Fundação Oswaldo Cruz. Instituto Oswaldo Cruz. Departamento de Virologia. Rio de Janeiro, RJ, Brasil.
Abstract
A survey on Plasmodium infection was carried out in gold mine camps located in the Brazilian Amazon. Antibody
against P. falciparum ring-infected erythrocyte surface antigen (RESA) was quantified by an enzyme-immunoassay
in order to assess P. falciparum exposure. Hepatitis B, a common infection in this area, was also investigated by
serologic markers. Among 520 sampled subjects, 517 (99.4%) admitted previous symptomatic malaria, 106 (20.4%)
had positive thick smears for malaria, 82.9% had HBV markers, and 7.1% were HBsAg positive. Anti-RESA titers
was significantly lower in HBV carriers than in people with resolved HBV infection suggesting that the anti-RESA
immune response could be supressed by HBV carrier status. Moreover, immunedeficient responses to both infections
may take place in some subjects causing concomitant lower anti-RESA response and incapacity to clear HBV.
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